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<blockquote data-quote="BrockW" data-source="post: 440812" data-attributes="member: 5910"><p>I can appreciate at that.</p><p></p><p>The analysis and research has been done and continues to be done. Most of which is out there for public consumption.</p><p></p><p>Yes, to some degree it is. But there is reports of this taking place in small patches of Wisconsin. And the population studies and personal accounts seem to kind of corroborate. Those big robust pops east of the Mississippi just seem more durable.</p><p></p><p></p><p>Wasn’t my intention, might’ve misunderstood what you were getting at.</p><p></p><p>Recorded high prevalence, significantly lowered individual annual survivability, landowner and game and fish reports of reduced age structure that could not be accounted for even when accounting for increased harvest. Sustained lowered populations in localized areas, seeing sick deer on the landscape.</p><p></p><p></p><p>Well, in the GPS collar population studies, that doesn’t seem to be statistically possible once high prevalence is reached. The analysis has been done several times on several herds, east and west. Again, some are more durable than others.</p><p></p><p>But they don’t all die the same week. Slow growth in prevalence, exponential growth curve towards the end. That’s generally what prevalence looks like over time. So population is taking hits slowly over sustained period of time. Some get worse than others.</p><p></p><p>First found in the wild in 1981….</p><p></p><p></p><p>Well I think, considering how long they’ve had the disease, there are places that got as bad or worse in a shorter time period or are on that trajectory.</p><p></p><p>See what I said about culling in the previous post.</p><p></p><p></p><p>Culling stopped CWD in the wild in New York.</p><p>Slowed spread for several years in Illinois (but again, after a while it doesn’t work anymore). I think there’s some other states that have been able to record drops in prevalence with increased mortality.</p><p></p><p>Again, see previous post for my thoughts on culling.</p><p></p><p></p><p>I don’t fear it and don’t mean to be an alarmist, like I stated previously. It’s never been shown to transmit to humans or cattle. That’s an important distinction. That is fact.</p><p></p><p>I guess I’m saying, from a personal standpoint, the more I understand these prion diseases and how prions work, the weirder they get. It’s actually a very fascinating EPI when you start thinking about it.</p><p></p><p></p><p>Yes, they ingested prions. So they implemented regulations and stopped producing and selling products for human consumption that had prions in it.</p><p></p><p>I sincerely don’t mean to be an alarmist. Truly. But I’m not kiddin, these prions are weird little buggers. When you sit down and really think about what they are, how they work, and what they do. It’s kind of crazy to think about!</p></blockquote><p></p>
[QUOTE="BrockW, post: 440812, member: 5910"] I can appreciate at that. The analysis and research has been done and continues to be done. Most of which is out there for public consumption. Yes, to some degree it is. But there is reports of this taking place in small patches of Wisconsin. And the population studies and personal accounts seem to kind of corroborate. Those big robust pops east of the Mississippi just seem more durable. Wasn’t my intention, might’ve misunderstood what you were getting at. Recorded high prevalence, significantly lowered individual annual survivability, landowner and game and fish reports of reduced age structure that could not be accounted for even when accounting for increased harvest. Sustained lowered populations in localized areas, seeing sick deer on the landscape. Well, in the GPS collar population studies, that doesn’t seem to be statistically possible once high prevalence is reached. The analysis has been done several times on several herds, east and west. Again, some are more durable than others. But they don’t all die the same week. Slow growth in prevalence, exponential growth curve towards the end. That’s generally what prevalence looks like over time. So population is taking hits slowly over sustained period of time. Some get worse than others. First found in the wild in 1981…. Well I think, considering how long they’ve had the disease, there are places that got as bad or worse in a shorter time period or are on that trajectory. See what I said about culling in the previous post. Culling stopped CWD in the wild in New York. Slowed spread for several years in Illinois (but again, after a while it doesn’t work anymore). I think there’s some other states that have been able to record drops in prevalence with increased mortality. Again, see previous post for my thoughts on culling. I don’t fear it and don’t mean to be an alarmist, like I stated previously. It’s never been shown to transmit to humans or cattle. That’s an important distinction. That is fact. I guess I’m saying, from a personal standpoint, the more I understand these prion diseases and how prions work, the weirder they get. It’s actually a very fascinating EPI when you start thinking about it. Yes, they ingested prions. So they implemented regulations and stopped producing and selling products for human consumption that had prions in it. I sincerely don’t mean to be an alarmist. Truly. But I’m not kiddin, these prions are weird little buggers. When you sit down and really think about what they are, how they work, and what they do. It’s kind of crazy to think about! [/QUOTE]
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